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Genetic analysis of the hypervariable region flanking the human insulin gene. Rotwein P, Yokoyama S, Didier DK, Chirgwin JM. Race-specific type 1 diabetes risk of HLA-DR7 haplotypes. HLA DR-DQ haplotypes and genotypes and type 1 diabetes risk: analysis of the type 1 diabetes genetics consortium families. 288(5465):505-11.Įrlich H, Valdes AM, Noble J, Carlson JA, Varney M, Concannon P, et al. A structural framework for deciphering the link between I-Ag7 and autoimmune diabetes. 329(6140):599-604.Ĭorper AL, Stratmann T, Apostolopoulos V, Scott CA, Garcia KC, Kang AS, et al. HLA-DQ beta gene contributes to susceptibility and resistance to insulin-dependent diabetes mellitus. 37(8):1113-9.Įrlich H, Valdes AM, Noble J, Carlson JA, Varney M, Concannon P, et al. Diabetes Epidemiology Research International Group. Geographic patterns of childhood insulin-dependent diabetes mellitus. 9(5):A355-65.ĭiabetes Epidemiology Research International Group. Concordance for islet autoimmunity among monozygotic twins. Redondo MJ, Jeffrey J, Fain PR, Eisenbarth GS, Orban T. Secondary attack rate of type 1 diabetes in Colorado families. Steck AK, Barriga KJ, Emery LM, Fiallo-Scharer RV, Gottlieb PA, Rewers MJ. A genome-wide search for human type 1 diabetes susceptibility genes. 2013 Apr 17.ĭavies JL, Kawaguchi Y, Bennett ST, Copeman JB, Cordell HJ, Pritchard LE, et al. Use of the Estimated Glucose Disposal Rate (eGDR) as a Measure of Insulin Resistance in an Urban Multiethnic Population With Type 1 Diabetes. April 25, 2013.Įpstein EJ, Osman JL, Cohen HW, Rajpathak SN, Lewis O, Crandall JP. Phenomenon of 'double diabetes' common among blacks. Effects of MCF2L2, ADIPOQ and SOX2 genetic polymorphisms on the development of nephropathy in type 1 Diabetes Mellitus. Vitamin d levels, microvascular complications, and mortality in type 1 diabetes. Joergensen C, Hovind P, Schmedes A, Parving HH, Rossing P. Vitamin D deficiency and coronary artery calcification in subjects with type 1 diabetes. Young KA, Snell-Bergeon JK, Naik RG, Hokanson JE, Tarullo D, Gottlieb PA, et al. High prevalence of capillary abnormalities in patients with diabetes and association with retinopathy. Genetics of the HLA region in the prediction of type 1 diabetes. Pancreatic volume and endocrine and exocrine functions in patients with diabetes. Philippe MF, Benabadji S, Barbot-Trystram L, et al. Prevalence of Type 1 diabetes autoantibodies (GAD and IA2) in Sardinian children and adolescents with autoimmune thyroiditis. Estimating the cost of type 1 diabetes in the U.S.: a propensity score matching method. Tao B, Pietropaolo M, Atkinson M, Schatz D, Taylor D. A Comparative Effectiveness Analysis of Three Continuous Glucose Monitors. Accessed: January 24, 2013.ĭamiano ER, El-Khatib FH, Zheng H, Nathan DM, Russell SJ. Continuous Glucose Monitoring: Navigator Beats Rival Devices. Standards of medical care in diabetes-2011. Development of Autoantibodies in the TrialNet Natural History Study. International Expert Committee report on the role of the A1C assay in the diagnosis of diabetes. Diagnosis and classification of diabetes mellitus. Advances in management of type 1 diabetes mellitus. Whether the diabetes is transient or chronic was also unknown.Īathira R, Jain V. The researchers suggested, however, that COVID-19 may induce diabetes by directly attacking pancreatic cells that express ACE2 receptors, that it may give rise to diabetes “through stress hyperglycemia resulting from the cytokine storm and alterations in glucose metabolism caused by infection,” or that COVID-19 may cause diabetes via the conversion of prediabetes to diabetes. The study could not specify the type or types of diabetes specifically related to COVID-19, with the report saying that the disease could be causing both type 1 and type 2 diabetes but through differing mechanisms. The investigators, using two US health claims databases, reported that pediatric patients with COVID-19 in the HealthVerity database were 31% percent more likely than other youth to receive a new diabetes diagnosis, while those in the IQVIA database were 166% more likely. A study from the US Centers for Disease Control and Prevention (CDC) indicates that SARS-CoV-2 infection increases the likelihood of diabetes developing in children under age 18 years, more than 30 days post infection.